Psoriasis is a Plaque-Psoriasis vulgaris autoimmune disease characterized by patches of abnormal Plaque-Psoriasis vulgaris. There are five main types of psoriasis: Psoriasis is generally thought to be a Plaque-Psoriasis vulgaris disease that is triggered by environmental factors. This suggests that genetic factors predispose to psoriasis. There is no Plaque-Psoriasis vulgaris for psoriasis; however, various treatments can help control the Plaque-Psoriasis vulgaris. These areas are called plaques and are most commonly found on the elbows, knees, scalp, and back.
It may be accompanied Plaque-Psoriasis vulgaris severe itching, swelling, and pain. It Plaque-Psoriasis vulgaris often the result of an exacerbation of unstable plaque psoriasis, particularly following the abrupt withdrawal of systemic glucocorticoids. Plaque-Psoriasis vulgaris include pustular, inverse, Plaque-Psoriasis vulgaris, guttate, oral, and seborrheic-like forms. Pustular Plaque-Psoriasis vulgaris appears as raised Plaque-Psoriasis vulgaris filled with noninfectious pus pustules.
Inverse psoriasis also known as flexural psoriasis appears as smooth, inflamed patches of skin. The patches frequently affect skin foldsparticularly around the genitals between the thigh and grointhe armpitsin the skin folds of an overweight abdomen known as Plaque-Psoriasis vulgarisbetween the buttocks in the intergluteal cleft, and under the breasts in the inframammary fold.
Heat, trauma, and infection are thought to play a role in the development of this atypical form of psoriasis.
Napkin psoriasis is a subtype of psoriasis common in infants characterized by red papules with silver Plaque-Psoriasis vulgaris in the diaper area that may Plaque-Psoriasis vulgaris to the torso or limbs. Guttate psoriasis is characterized by numerous small, scaly, red or pink, droplet-like lesions papules.
These numerous spots of psoriasis appear over large areas Plaque-Psoriasis vulgaris the body, primarily the trunk, but also the limbs Plaque-Psoriasis vulgaris scalp.
Guttate psoriasis is often triggered by a streptococcal infection, typically streptococcal pharyngitis. Psoriasis in the mouth is Plaque-Psoriasis vulgaris rare,  in contrast to Plaque-Psoriasis vulgaris planusanother common papulosquamous disorder that Plaque-Psoriasis vulgaris involves both the skin and mouth.
When psoriasis Plaque-Psoriasis vulgaris the oral mucosa the lining of the mouthit may be asymptomatic,  Plaque-Psoriasis vulgaris it may appear as white or grey-yellow plaques. The microscopic appearance of oral mucosa affected by geographic tongue migratory stomatitis is very similar to the appearance of psoriasis.
Seborrheic-like psoriasis is a common form of psoriasis with clinical aspects of psoriasis and seborrheic dermatitisand it may be difficult to distinguish from the latter. This form of psoriasis typically manifests as red plaques Plaque-Psoriasis vulgaris greasy scales in areas of higher sebum production such as the scalpforeheadskin folds next to Plaque-Psoriasis vulgaris noseskin surrounding the mouth, skin on the chest above the sternumPlaque-Psoriasis vulgaris in skin folds.
Psoriatic arthritis is a form of chronic inflammatory arthritis that has a highly variable clinical presentation and frequently occurs in association with skin and nail psoriasis. This can result in a sausage-shaped swelling Plaque-Psoriasis vulgaris the fingers and toes known as dactylitis.
Psoriasis Plaque-Psoriasis vulgaris affect the nails and produces a Plaque-Psoriasis vulgaris of changes in the appearance of finger and toe nails. In addition to the appearance and distribution of the rash, specific medical learn more here may be used by medical practitioners to assist with diagnosis. These may include Auspitz's sign Plaque-Psoriasis vulgaris bleeding when scale is removedKoebner phenomenon psoriatic skin lesions induced by trauma to the skin Plaque-Psoriasis vulgaris,  and itching Plaque-Psoriasis vulgaris pain localized to papules and plaques.
Around Plaque-Psoriasis vulgaris of people with psoriasis report a family history of Plaque-Psoriasis vulgaris disease, and researchers have identified genetic loci associated Plaque-Psoriasis vulgaris the condition. These findings suggest both a genetic susceptibility and an environmental response in developing psoriasis. Psoriasis has a strong hereditary component, and many genes are associated with it, but it is unclear how those genes work together.
Most of the identified genes relate to the immune system, particularly the major histocompatibility complex MHC and T cells. Genetic studies are valuable due to their ability to identify molecular mechanisms and pathways for further study and potential drug targets. Classic genome-wide linkage http://wirtschafts-indikatoren.de/corofacecet/john-psoriasis-peganolechenie.php has identified Plaque-Psoriasis vulgaris loci on different chromosomes associated with psoriasis.
Within those loci are genes on pathways that lead to inflammation. Certain variations mutations of those genes are commonly Psoriasis Foto oder Flechte in psoriasis. Some of these genes express inflammatory signal proteins, which affect cells in the immune system that are also involved in psoriasis.
Some of these genes are also involved in other autoimmune diseases. Two major immune system genes under investigation are interleukin subunit beta IL12B on chromosome 5qwhich expresses Plaque-Psoriasis vulgaris and IL23R on chromosome 1p, which expresses the interleukin receptor, and is involved in T cell differentiation.
Interleukin receptor and IL12B have both been strongly linked with psoriasis. A rare here in the gene encoding for the CARD14 protein plus an environmental trigger was enough Plaque-Psoriasis vulgaris cause plaque psoriasis the most Plaque-Psoriasis vulgaris form of psoriasis.
Conditions reported as worsening the disease include chronic infections, stress, and changes in Plaque-Psoriasis vulgaris and climate. The rate of psoriasis in HIV-positive individuals is comparable to that of HIV-negative individuals, however, psoriasis tends to be Plaque-Psoriasis vulgaris severe in people welche Art von Fisch können Sie bei Psoriasis with HIV.
Psoriasis has been described as occurring after strep throatand may be worsened by skin or gut colonization with Staphylococcus aureusMalasseziaand Candida albicans. Drug-induced psoriasis may occur with beta blockers Plaque-Psoriasis vulgaris antimalarial medications non-steroidal anti-inflammatory drugs terbinafinecalcium channel blockerscaptoprilglyburidegranulocyte colony-stimulating factor interleukinsinterferons lipid-lowering drugsPlaque-Psoriasis vulgaris Psoriasis is characterized by an abnormally excessive and rapid growth of the epidermal layer of the skin.
Plaque-Psoriasis vulgaris mutations of proteins involved in the skin's ability to function as a barrier have been identified Plaque-Psoriasis vulgaris markers of susceptibility for the development of psoriasis. Dendritic cells bridge the innate immune system Plaque-Psoriasis vulgaris adaptive immune system. They are increased in psoriatic lesions  and induce the proliferation of T cells and type 1 helper T cells Th1. A diagnosis of psoriasis is usually based on the appearance of the skin.
Skin characteristics typical for Plaque-Psoriasis vulgaris are scaly, erythematous plaques, papules, or patches of skin Plaque-Psoriasis vulgaris may Plaque-Psoriasis vulgaris painful Plaque-Psoriasis vulgaris itch.
If the clinical diagnosis is uncertain, a skin biopsy or scraping may Plaque-Psoriasis vulgaris performed to rule out Plaque-Psoriasis vulgaris disorders and Plaque-Psoriasis vulgaris confirm the diagnosis. Skin Plaque-Psoriasis vulgaris a biopsy will show clubbed epidermal projections that interdigitate with dermis on microscopy.
Epidermal thickening is another characteristic histologic finding of psoriasis lesions. Unlike their mature counterparts, these superficial cells keep Plaque-Psoriasis vulgaris nucleus. Psoriasis is classified as a papulosquamous disorder and Plaque-Psoriasis vulgaris most commonly subdivided into different Plaque-Psoriasis vulgaris based on histological characteristics.
Each form has a dedicated ICD Plaque-Psoriasis vulgaris. Another classification scheme considers genetic and demographic factors. Plaque-Psoriasis vulgaris 1 has a positive family history, starts before the age of 40, and is associated with the human leukocyte antigenHLA-Cw6. Conversely, type 2 does not show a family history, presents after Plaque-Psoriasis vulgaris 40, and is not associated with HLA-Cw6.
Plaque-Psoriasis vulgaris classification of psoriasis as an autoimmune disease has sparked considerable debate. Researchers have proposed differing descriptions of psoriasis and psoriatic arthritis; some authors have classified them as autoimmune diseases  Plaque-Psoriasis vulgaris  while others have classified them as distinct from autoimmune diseases and http://wirtschafts-indikatoren.de/corofacecet/kreis-schuppenflechte.php to them as immune-mediated Plaque-Psoriasis vulgaris diseases.
There is no consensus about how to classify Plaque-Psoriasis vulgaris severity of psoriasis. The DLQI score Plaque-Psoriasis vulgaris from 0 minimal impairment to 30 maximal impairment and is calculated with each answer being assigned 0—3 points with higher scores indicating greater social or occupational impairment. The psoriasis area severity index PASI is the most widely used measurement tool for psoriasis. PASI assesses the severity of lesions and the area affected and combines these two factors into a single score from 0 no disease to 72 maximal disease.
While no cure is available for psoriasis,  many treatment options exist. Topical agents are typically used for mild disease, phototherapy for moderate disease, and systemic agents for severe disease. Topical corticosteroid preparations are the most effective agents when used continuously for 8 weeks; retinoids and coal tar were found to be of limited benefit and may be no better than placebo.
Plaque-Psoriasis vulgaris D analogues such as paricalcitol were found to be superior to placebo. Combination therapy with vitamin D and a corticosteroid was superior to either treatment alone and vitamin D was found to be superior to coal tar for chronic plaque psoriasis. For psoriasis of the scalp, a review found dual therapy vitamin D Plaque-Psoriasis vulgaris and topical corticosteroids or corticosteroid monotherapy to be more effective and safer than topical vitamin D analogues alone.
Moisturizers and emollients such as mineral oilpetroleum jellycalcipotrioland decubal an oil-in-water emollient were found to increase the clearance of psoriatic plaques. Emollients have been shown to be even more effective at clearing psoriatic plaques when combined with phototherapy. Plaque-Psoriasis vulgaris emollient salicylic acid is structurally similar to para-aminobenzoic acid PABAcommonly found in sunscreen, and is known to interfere with phototherapy in psoriasis.
Coconut oilwhen used as an emollient in psoriasis, has been found to decrease plaque clearance with phototherapy. Ointment and creams containing coal tardithranolcorticosteroids i. The use of the finger tip unit may be helpful in guiding how much topical treatment to use.
Vitamin D analogues may be Plaque-Psoriasis vulgaris with steroids; however, alone have a higher rate of side effects. Another topical therapy used to treat psoriasis is a form Plaque-Psoriasis vulgaris balneotherapywhich involves Plaque-Psoriasis vulgaris baths in the Dead Sea. This is usually done for four weeks with the benefit attributed to sun exposure and specifically UVB light. This is cost-effective and it has been propagated as an effective way to treat psoriasis without medication.
Phototherapy in the form of sunlight has long been used for psoriasis. The UVB lamps should have a timer that will turn off the lamp when the time ends. The amount of light Plaque-Psoriasis vulgaris is determined by a person's skin type. One of the problems with clinical phototherapy is the difficulty many patients have gaining access to a facility.
Indoor tanning resources are almost ubiquitous today and could be considered as a means for patients to get UV exposure when dermatologist provided phototherapy is not available. However, a concern with the use of Plaque-Psoriasis vulgaris tanning is that tanning beds that primarily emit UVA might not effectively treat psoriasis.
One study found that plaque psoriasis is responsive to erythemogenic doses of either UVA or UVB, as exposure to either can cause dissipation of Fufaev Psoriasis rein Lotion plaques.
It does require more energy to reach erythemogenic dosing with UVA. UV light therapies all have risks; tanning beds are no exception, particularly in the link between UV light and the increased chance of skin cancer. There are increased Plaque-Psoriasis vulgaris of Plaque-Psoriasis vulgaris, squamous cell and basal cell carcinomas; younger psoriasis patients, particularly those under age 35, are at increased check this out from melanoma from UV light Plaque-Psoriasis vulgaris. A review of studies recommends that people who are susceptible Plaque-Psoriasis vulgaris skin cancers exercise caution when using UV light therapy as a treatment.
Plaque-Psoriasis vulgaris type of phototherapy is useful in the treatment of psoriasis because the formation of these dimers interferes with the cell cycle and stops it. The interruption of the cell cycle induced by NBUVB opposes the characteristic rapid division of skin cells seen in psoriasis. The most common short-term side effect of this form of phototherapy is redness of the skin; less common side effects of NBUVB phototherapy are itching and blistering of the treated skin, irritation of the eyes in the form of conjunctival inflammation or inflammation of the corneaor cold sores due to reactivation of the herpes simplex virus in the skin surrounding the lips.
Eye protection is usually given during phototherapy treatments. The mechanism of action of PUVA Plaque-Psoriasis vulgaris unknown, but probably involves activation of psoralen by UVA light, which Plaque-Psoriasis vulgaris the abnormally rapid production of the cells in psoriatic skin. There are multiple mechanisms of action associated with PUVA, including effects Psoriasis im Kopf als Abstrich the more info immune system.
PUVA is associated with nauseaheadachefatigueburning, and itching.
Psoriasis - Wikipedia
Man unterscheidet verschiedene Formen der Psoriasis, die in ihrem Erscheinungsbild und ihrer Plaque-Psoriasis vulgaris Schmerzen in den Ellenbogengelenken und Psoriasis voneinander Plaque-Psoriasis vulgaris können. Die bei weitem dominierende Erkrankungsform ist die Psoriasis vulgaris Plaque-Psoriasis 1.
Typische Nagelsymptome zeigt die Abbildung 3— Zur Diagnostik der Plaque-Psoriasis. Ich stimme zu, dass diese Seite Cookies für Analysen und Funktionen verwendet. Klinisches Bild der Plaque-Psoriasis Man unterscheidet verschiedene Formen der Psoriasis, die in ihrem Erscheinungsbild Plaque-Psoriasis vulgaris ihrer Ausprägung deutlich voneinander differieren können.
Neben den Hautläsionen treten bei vielen Patienten auch Nagelveränderungen 3, 4, 5 auf. Typische Nagelsymptome Psoriasis Heilung die Abbildung 3—5: Das klinische Spektrum der Nagelpsoriasis. S3-Leitlinie zur Therapie Plaque-Psoriasis vulgaris Psoriasis vulgaris.
JDDG ; Plaque-Psoriasis vulgaris Suppl 2: Quality of psoriasis care in Germany — results Plaque-Psoriasis vulgaris the national study PsoHealth Nail psoriasis in Germany: Psoriasis — neue Erkenntnisse zur Pathogenese und Therapie. Dtsch Ärztebl Plaque-Psoriasis vulgaris Schweiz Med Forum ; 6: Rheumatische Gelenkerkrankungen Rheumatoide Arthritis. Chronisch-entzündliche Darmerkrankungen Morbus Crohn.
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Psoriasis Vulgaris Plaque | Psoriasis Disease ☼ It typically affects the outside of the elbows, knees or scalp, though it can appear ★★ .
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Picture of Psoriasis Vulgaris Plaque Psoriasis vulgaris plaque, or Koebner phenomenon, is the characteristic skin rash of psoriasis. The photo depicts Koebner phenomenon as .
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Psoriasis Vulgaris Plaque | Psoriasis Disease ☼ It typically affects the outside of the elbows, knees or scalp, though it can appear ★★ .